Melinda K. Duncan, Ph.D.
Professor
Graduate Program Director
Duncan
Phone: (302) 831-0533
Fax: (302) 831-2281
Email: duncanm@udel.edu
Office: 327 Wolf Hall
Lab: 266 Wolf Hall
Address:
Department of Biological Sciences
Wolf Hall
University of Delaware
Newark, DE 19716
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Education
- B.S. - Lafayette College
- Ph.D. - The University of Medicine and Dentistry of New Jersey, Robert Wood Johnson Medical School and Rutgers the State University of New Jersey
- Postdoctoral - The National Eye Institute, The National Institutes of Health
Teaching
- BISC 415/615; ANSC 415/615 - Vertebrate Developmental Biology
- BISC 654* - Biochemical Genetics
- BISC 827** - Graduate Research Seminar
*Course web site available through Sakai
**Course web site available through MyCourses
Research Interests

Lens isolated from an adult cow
The vertebrate lens is a remarkable tissue that has many evolutionary adaptations that allow it to remain transparent throughout life. Further, its cell biology is quite distinct from that of other tissues which results in it being the only epithelial organ which never develops clinically relevant cancers. Aging, ocular injury, genetic alterations or systemic diseases such as diabetes can lead to the loss of lens transparency, or cataract. Cataracts are the predominant cause of blindness worldwide and their incidence is increasing as life expectancies increase. Over the past thirty years, robust techniques to surgically treat cataract have been developed, greatly reducing cataract-related blindness in western countries. In the United States, cataract removal is the most common surgical procedure performed, representing over 2.5% of the total Medicare budge. While the short term visual outcome for patients undergoing cataract surgery is often excellent, 10-25% of operated eyes subsequently develop posterior capsular opacification (PCO), a condition often referred to as secondary cataract which requires additional treatment and can lead to poor vision in the long term.
My laboratory focuses on understanding the transcriptional control of cell differentiation in the lens and the role of integrins and extracellular matrix proteins in regulating normal lens biology and their alterations in cataracts and PCO.
Current Projects
- Transcriptional control of lens fiber cell differentiation - The differentiation of lens fiber cells is marked by a major reorganization of cell structure and drastic changes in gene expression. βB1-crystallin, one of the genes whose expression is first detected coincident with the morphological differentiation of lens fiber cells, is often used as a molecular marker for this process. We have isolated the complete promoter of this gene and identified some of the cis-elements and transcription factors responsible for its function. We are studying the biochemistry of these transcription factors, particularly how their expression and function is controlled by fiber cell differentiation signals arising outside of the lens. Further, since many of the transcription factors that control βB1-crystallin expression are also expressed in the lens epithelium, we are also studying the influence of other regulators of gene expression, such as chromatin remodeling enzymes, on lens fiber cell differentiation.
- The influence of the lens capsule on lens development and function - The lens is a true epithelium with the basal surface of every transcriptionally active cell attached to a modified basement membrane, the lens capsule. While numerous studies have suggested that extracellular matrix molecules can influence lens cell biology, little is known about either the molecular composition of the capsule or the nature of the receptors present on lens cells that mediate lens cell/lens capsule interactions. We have previously shown that collagen IV is present in the lens capsule and that the molecular composition of the collagen IV network changes during lens development. We are now investigating the functional significance of these changes both in vivo and in vitro as well as the molecular mechanisms by which lens cells could detect a changing collagen IV network.
Research Group
- Rebecca Helton, Ph.D. - Postdoctoral Fellow (Ph.D., University of Delaware). Development of conditional null mice.
- Vladimir Simirskii, Ph.D. - Postdoctoral Fellow (Ph.D., Russian Academy of Science, Russia). The lens capsule and lens development.
- Yan Wang, M.D. - Laboratory Researcher (M.D., China Medical University, China). Mammalian lens regeneration.
- Dylan Audette, B.A. - Graduate Student (B.A., University of Delaware). Prox1 function in lens.
- Brian Danysh, B.A. - Graduate Student (B.A., University of Delaware). Role of the lens capsule in lens biology.
- Zeynep Firtina, B.S. - Graduate Student (B.S., Sabanci University, Turkey). Unfolded protein response in cataract development.
- Abby Grabitz, B.S. - Graduate Student (B.S., Alma College). Lens development.
- Fahmy Mamuya, B.S. - Graduate Student (B.S., Grand Valley State University). Integrin functions in the lens.
- David Scheiblin, B.A. - Graduate Student (B.A., University of Delaware). Integrin function in lens fibers.
- Matthew King - Junior Biology major. Fibronectin in lens.
- Robert Sheehan - Junior Quantitative Biology major. Histone dynamics during fiber cell differentiation.
- Jamie Stull - Junior Cellular and Molecular Biology major. Unfolded protein response induced by mutations in ECM genes.
Selected Publications
- Danysh BP, Duncan MK. The lens capsule. Exp Eye Res. 2009;88(2):151–164.
- Chen X, Patel TP, Simirskii VI, Duncan MK. PCNA interacts with Prox1 and represses its transcriptional activity. Mol Vis. 2008;14:2076–2086.
- Chen X, Taube JR, Simirskii VI, Patel TP, Duncan MK. Dual roles for Prox1 in the regulation of the chicken betaB1-crystallin promoter. Invest Ophthalmol Vis Sci. 2008;49(4):1542–1552.
- Danysh BP, Czymmek KJ, Olurin PT, Sivak JG, Duncan MK. Contributions of mouse genetic background and age on anterior lens capsule thickness. Anat Rec (Hoboken). 2008;291(12):1619–1627.
- Lucey MM, Wang Y, Bustin M, Duncan MK. Differential expression of the HMGN family of chromatin proteins during ocular development. Gene Expr Patterns. 2008;8(6):433–437.
- Naik MU, Naik TU, Suckow AT, Duncan MK, Naik UP. Attenuation of junctional adhesion molecule-A is a contributing factor for breast cancer cell invasion. Cancer Res. 2008;68(7):2194–2203.
- Ramalingam A, Duhadaway JB, Sutanto-Ward E, et al. Bin3 deletion causes cataracts and increased susceptibility to lymphoma during aging. Cancer Res. 2008;68(6):1683–1690.
- Cvekl A, Duncan MK. Genetic and epigenetic mechanisms of gene regulation during lens development. Prog Retin Eye Res. 2007;26(6):555–597.
- Duprey KM, Robinson KM, Wang Y, Taube JR, Duncan MK. Subfertility in mice harboring a mutation in betaB2-crystallin. Mol Vis. 2007;13:366–373.
- Kang LI, Wang Y, Suckow AT, et al. Deletion of JAM-A causes morphological defects in the corneal epithelium. Int J Biochem Cell Biol. 2007;39(3):576–585.
- Simirskii VN, Wang Y, Duncan MK. Conditional deletion of beta1-integrin from the developing lens leads to loss of the lens epithelial phenotype. Dev Biol. 2007;306(2):658–668.
- Chen X, Patel TP, Cain WJ, Duncan MK. Production of monoclonal antibodies against Prox1. Hybridoma (Larchmt). 2006;25(1):27–33.
- Huang KM, Wu J, Duncan MK, et al. Xcat, a novel mouse model for Nance-Horan syndrome inhibits expression of the cytoplasmic-targeted Nhs1 isoform. Hum Mol Genet. 2006;15(2):319–327.
- Simirskii VN, Lee RS, Wawrousek EF, Duncan MK. Inbred FVB/N mice are mutant at the cp49/Bfsp2 locus and lack beaded filament proteins in the lens. Invest Ophthalmol Vis Sci. 2006;47(11):4931–4934.
- Castellini M, Wolf LV, Chauhan BK, et al. Palm is expressed in both developing and adult mouse lens and retina. BMC Ophthalmol. 2005;5:14.
- Collinge JE, Simirskii VN, Duncan MK. Expression of tissue plasminogen activator during eye development. Exp Eye Res. 2005;81(1):90–96.
- Parris JJ, Cooke VG, Skarnes WC, Duncan MK, Naik UP. JAM-A expression during embryonic development. Dev Dyn. 2005;233(4):1517–1524.
- Cui W, Ning J, Naik UP, Duncan MK. OptiRNAi, an RNAi design tool. Comput Methods Programs Biomed. 2004;75(1):67–73.
- Cui W, Tomarev SI, Piatigorsky J, Chepelinsky AB, Duncan MK. Mafs, Prox1, and Pax6 can regulate chicken betaB1-crystallin gene expression. J Biol Chem. 2004;279(12):11088–11095.
- Duncan MK, Cvekl A, Kantorow M, Piatigorsky J. Lens crystallins in Development of the Ocular Lens. Frank J. Lovicu, Michael L. Robinson, eds. Cambridge University Press; 2004.
- Duncan MK, Xie L, David LL, et al. Ectopic Pax6 expression disturbs lens fiber cell differentiation. Invest Ophthalmol Vis Sci. 2004;45(10):3589–3598.
- Vasavada TK, DiAngelo JR, Duncan MK. Developmental expression of Pop1/Bves. J Histochem Cytochem. 2004;52(3):371–377.
- West KL, Castellini MA, Duncan MK, Bustin M. Chromosomal proteins HMGN3a and HMGN3b regulate the expression of glycine transporter 1. Mol Cell Biol. 2004;24(9):3747–3756.
- Wilmarth PA, Taube JR, Riviere MA, Duncan MK, David LL. Proteomic and sequence analysis of chicken lens crystallins reveals alternate splicing and translational forms of beta B2 and beta A2 crystallins. Invest Ophthalmol Vis Sci. 2004;45(8):2705–2715.
- Azuma M, Tamada Y, Kanaami S, et al. Differential influence of proteolysis by calpain 2 and Lp82 on in vitro precipitation of mouse lens crystallins. Biochem Biophys Res Commun. 2003;307(3):558–563.